Intensive systolic blood pressure (BP) lowering (<140 mm Hg) after successful endovascular thrombectomy significantly increases the risk of acute kidney injury (AKI) compared to conventional BP targets (140-180 mm Hg).
Most AKI events observed were mild (stage 1), yet they were associated with markedly worse neurological outcomes, including lower functional independence and higher mortality at 3 months.
AKI after thrombectomy represents a marker of systemic hemodynamic vulnerability and indicates the need for cautious post-procedure BP management.
