Highlights
Catheter ablation serves as a potent bailout intervention for incessant ventricular tachycardia (VT) in the early phase of acute myocardial infarction (AMI) when conventional pharmacological and hemodynamic supports are exhausted.
Contrary to previous assumptions of purely ischemic triggers, intracardiac mapping confirms that scar-related reentry within the territory of the culprit artery is the primary driver of refractory VT in early AMI.
